On November 22, 2013, 23andMe, Inc. was forced to stop allowing its customers to access their health reports on genetic information because these genomic reports may be unreliable and misinterpreted, as expressed by the Food and Drug Administration (FDA). The FDA's main concern is that customers could use their genomic information to take unprecedented actions that could expose them to health risks by going to extremes to correct this allele, which could lead to higher rates. high rates of diseases when there is not even a risk of having this allele, or lead them to underestimate their state of health, if consumers interpreted that they did not have a gene coding for obesity . The FDA restrictions were correct because customers should not rely on these health reports to determine whether they have a genetic allele that predisposes them to obesity, because genetic influences do not determine to a large extent whether a individual is more susceptible to obesity during childhood and adolescence. With further understanding, the FTO gene associated with BMI does not predispose the individual to a higher risk of susceptibility to obesity, since the FTO locus only explains 1% of the total heritability of the BMI and that the role of epigenetics largely eclipses the influence of genetics. , genome-wide association studies (GWAS) have been used to identify the underlying genomic basis of diseases, including obesity. But the slow progress and limited conclusions that can be drawn from the study of the identified 32-GWAS loci prove that the identification of an obesity risk allele at one of these loci or at the FTO gene is weak and does not have enough weight to prove it. that the individual is indeed predisposed to an obesity phenotype. Over 61 generations... middle of paper ...... stability is a proportion and is still undeniably affected by environmental factors, and this is where epigenetics comes in. Although genetics plays a role in BMI variation in individuals, this does not mean that our body weight is genetically predetermined, because the "obesogenic" environment we find ourselves in, daily calorie intake and amount of physical activity all play a role. a role in determining our phenotype. The heritability of BMI is balanced by a combination of all these factors. Thus, 23andMe, Inc. should not have the right to give its customers access to their genome health reports, because having an obesity risk allele in the FTO gene does not necessarily put them at risk at a higher risk of being predisposed to obesity, and could lead them to extremes in fighting this disease, while its absence, the risk allele could relax their precautions in the fight against obesity.